Cite

1. Arnardottir HH, Dalli J, Norling LV, Colas RA et al. Resolvin D3 is dysregulated in arthritis and reduces arthritic inflammation. J Immunol 2016; 197(6):2362-2368.10.4049/jimmunol.1502268501100627534559Search in Google Scholar

2. Barden AE, Moghaddami M, Mas E et al. Specialised pro-resolving mediators of inflammation in inflammatory arthritis. Prostaglandins Leukot Essent Fatty Acids 2016; 107:24-29.10.1016/j.plefa.2016.03.00427033423Search in Google Scholar

3. Benvenuti M, An T, Amaro E et al. Double-Edged Sword: Musculoskeletal Infection Provoked Acute Phase Response in Children. Orthop Clin North Am. 2017; 48(2):181-197.10.1016/j.ocl.2016.12.00928336041Search in Google Scholar

4. Buckley CD, Gilroy DW, Serhan CN. Pro-Resolving lipid mediators and mechanisms in the resolution of acute inflammation. Immunity. 2014; 40(3):315-327.10.1016/j.immuni.2014.02.009400495724656045Search in Google Scholar

5. Chiang N, Serhan CN. Structural Elucidation and Physiologic Functions of Specialized Pro-Resolving Mediators and Their Receptors. Mol Aspects Med. 2017; 58:114-129.10.1016/j.mam.2017.03.005562360128336292Search in Google Scholar

6. Cho JG, Lee A, Chang W et al. Endothelial to Mesenchymal Transition Represents a Key Link in the Interaction between Inflammation and Endothelial Dysfunction. Front Immunol. 2018; 9:294.10.3389/fimmu.2018.00294582619729515588Search in Google Scholar

7. Colville-Nash PR, Scott DL. Angiogenesis and rheumatoid arthritis: pathogenic and therapeutic implications. Annals of the Rheumatic Diseases 1992; 51:919-925.10.1136/ard.51.7.91910047841378718Search in Google Scholar

8. Costa C, Incio J, Soares R. Angiogenesis and chronic inflammation: cause or consequence? Angiogenesis. 2007; 10(3):149-66.10.1007/s10456-007-9074-017457680Search in Google Scholar

9. Díaz-Flores L, Gutiérrez R, García-Suárez MP et al. Morphofunctional basis of the different types of angio-genesis and formation of postnatal angiogenesis-related secondary structures. Madrid Histol Histopathol 2017; 32:1239-1279.Search in Google Scholar

10. DiPietro LA. Angiogenesis and wound repair: when enough is enough. Jornal of leukocyte biology 2016; 100(5), 979-984.10.1189/jlb.4MR0316-102R660806627406995Search in Google Scholar

11. Elshabrawy HA, Chen Z, Volin MV et al. The Pathogenic Role of Angiogenesis in Rheumatoid Arthritis. Angiogenesis. 2015; 18(4):433-448.10.1007/s10456-015-9477-2487988126198292Search in Google Scholar

12. Fredman G, Tabas I. Boosting Inflammation Resolution in Atherosclerosis. Am J Pathol 2017; 187(6):1211-1221.10.1016/j.ajpath.2017.01.018Search in Google Scholar

13. Majno G. Chronic Inflammation. Am J Pathol 1998; 153(4):1035-1039.10.1016/S0002-9440(10)65648-9Search in Google Scholar

14. Moshage H. Cytokines and the hepatic acute phase response. Journal of Pathology 1997; vol. 181:257-266.10.1002/(SICI)1096-9896(199703)181:3<257::AID-PATH756>3.0.CO;2-USearch in Google Scholar

15. Nathan C, Ding A. Review: Nonresolving Inflammation. Cell 2010; 140, 871-882.10.1016/j.cell.2010.02.029Search in Google Scholar

16. Orlowsky EW, Kraus VB. The Role of Innate Immunity in Osteoarthritis: When Our First Line of Defense Goes on the Offensive. J Rheumatol 2015; 42(3):363-371.10.3899/jrheum.140382Search in Google Scholar

17. Ridiandries A, Tan JTM, Bursill CA. The Role of Chemokines in Wound Healing. Int J Mol Sci 2018; 19(10), 3217.10.3390/ijms19103217Search in Google Scholar

18. Roemer FW. Presence of MRI-detected joint effusion and synovitis increases the risk of cartilage loss in knees without osteoarthritis at 30-month follow-up: the MOST study. Ann Rheum Dis 2011; 70(10):1804-1809.10.1136/ard.2011.150243Search in Google Scholar

19. Scanzello CR, Plaas A, Crow MK. Innate immune system activation in osteoarthritis: is osteoarthritis a chronic wound? Curr Opin Rheumatol 2008; 20(5):565-72.Search in Google Scholar

20. Scheibner KA, Lutz MA, Boodoo S et al. Hyaluronan Fragments Act as an Endogenous Danger Signal by Engaging TLR2. J Immunol 2006; 177 (2) 1272-1281.10.4049/jimmunol.177.2.1272Search in Google Scholar

21. Senger DR, Davis GE. Angiogenesis Cold Spring Harb Perspect. Biol 2011; 3(8):a005090.10.1101/cshperspect.a005090Search in Google Scholar

22. Serhan C.N. Novel Pro-Resolving Lipid Mediators in Inflammation Are Leads for Resolution Physiology. Nature 2014; 510(7503):92-101.10.1038/nature13479Search in Google Scholar

23. Serhan CN, Yacoubian S, Yang R. Anti-Inflammatory and Pro-Resolving Lipid Mediators. Annu Rev Pathol 2008; 3:279-312.10.1146/annurev.pathmechdis.3.121806.151409Search in Google Scholar

24. Serhan CN, Chiang N, Dalli J. The Resolution Code of Acute Inflammation: Novel Pro-Resolving Lipid Mediators in Resolution. Semin Immunol 2015; 27(3):200-215.10.1016/j.smim.2015.03.004Search in Google Scholar

25. Serhan CN. Treating inflammation and infection in the 21st century: new hints from decoding resolution mediators and mechanisms. FASEB J 2017; 31(4):1273-1288.10.1096/fj.201601222RSearch in Google Scholar

26. Thurston G, Murphy TJ, Baluk P et al. Angiogenesis in Mice with Chronic Airway Inflammation. Am J Pathol 1998; 153(4):1099-1112.10.1016/S0002-9440(10)65654-4Search in Google Scholar

27. Uddin M, Levy BD. Resolvins: Natural Agonists for Resolution of Pulmonary Inflammation. Prog Lipid Res 2011n; 50(1):75-88.10.1016/j.plipres.2010.09.002301213920887750Search in Google Scholar

28. Van Caam A, Vonk M, van den Hoogen F et al. Unraveling SSc Pathophysiology; The Myofibroblast. Frontiers in Immunology 2018; 9(2452):1-22.10.3389/fimmu.2018.02452624295030483246Search in Google Scholar

eISSN:
2335-075X
ISSN:
1820-8665
Language:
English
Publication timeframe:
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Journal Subjects:
Medicine, Clinical Medicine, other