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Fig. 1
Weight gain of infected ducks. **P < 0.01 and ***P < 0.001
Fig. 2
Gross lesions of infected ducks. They were severe thymic atrophy, a mild to moderately atrophied spleen and bursa of Fabricius, pale bone marrow and spleen, and hepatic necrosis in the GPV and DuCV coinfected group. Scale bar = 1 cm
Fig. 3
Histological lesions of infected ducks at 30 dpi (200×). A. Lesions of the liver, kidneys, and duodenum. Extensive hepatocellular vacuolar degeneration, necrosis, and lymphocyte infiltration in the liver, severe degeneration, necrosis, exfoliation, and heterophilic granulocyte infiltration in renal tubular epithelial cells, and necrosis and exfoliation, haemorrhage, glandular tube atrophy, and lymphocyte infiltration in the duodenal villi are visible. B. Lesions of immune organs. Lymphocyte loss and haemorrhage in the spleen, infiltrating heterotropic granulocytes, thymic corpuscle disintegration, loss of lymphocytes in the cortex and medulla of the bursa, and histiocyte loss in the bone marrow are visible
Fig. 4
Dynamic changes of GPV and DuCV loads in tissues of experimentally infected ducks. A. DuCV loads at 10 dpi; B. DuCV loads at 20 dpi; C. DuCV loads at 30 dpi; D. GPV loads at 10 dpi; E. GPV loads at 20 dpi; F. GPV loads at 30 dpi. *P < 0.05, **P < 0.01, and ***P < 0.001