The incidence of dysrhythmias after administration of the antipsychotic olanzapine

[1] Anastassion X. Psychosis : Causes, Diagnosis and Treatment. New York : Nova Science Publishers. 2012Search in Google Scholar

[2] Bresee LC, Majmdar SR, Patten SB, et al. Prevalence of cardiovascular risk factors and disease in people with schizophrenia: A population-based study. In Schizophrenia Research. 2010; 117(1):75-8210.1016/j.schres.2009.12.01620080392Search in Google Scholar

[3] Fossa AA. The impact of varying autonomic states on the dynamic beat-to-beat QT-RR and QT-TQ interval relationships. Br J Pharmacol. 2008,154:1508-151510.1038/bjp.2008.123249208918663381Search in Google Scholar

[4] Gintant GA, Gallacher DJ, Pugsley MK. The ‘overly-sensitive’ heart: Sodium channel block and QRS interval prolongation. Br J Pharmacol. 2011,164: 254-25910.1111/j.1476-5381.2011.01433.x317440621488862Search in Google Scholar

[5] Jones M, Campbell G, Patel D, et al. Risk of Mortality (including Sudden Cardiac Death) and Major Cardiovascular Events in Users of Olanzapine and Other Antipsychotics. Cardiovascular Psychiatry and Neurology. 2013, s. 1-1310.1155/2013/647476387659224416588Search in Google Scholar

[6] Lee SH, Kim HR, Han RX, et al. Cardiovascular risk assessment of atypical antipsychotic drugs in a zebrafish model. J. Appl. Toxicol. 2013; 33(6):466-470Search in Google Scholar

[7] Moss AJ. The QT interval and torsade de pointes. Drug Saf. 1999, 21(1)10.2165/00002018-199921001-0000210597863Search in Google Scholar

[8] Pasterbak B, Svanstrom H, Ranthe MF, et al. Atypical Antipsychotics Olanzapine, Quetiapine, and Risperidone and Risk of Acute Major Cardiovascular Events in Young and Middle-Aged Adults: A Nationwide Register-Based Cohort Study in Denmark. CNS Drugs. 2014; 963-97310.1007/s40263-014-0176-024895158Search in Google Scholar

[9] Shafti SS, Jahromi P. Olanzapine induced Q-Tc shortening. Ther Adv Psychopharmacol. 2014; 4(6):240-24610.1177/2045125314546484425798425489475Search in Google Scholar

[10] Silvestre JS, O´Neill MF, Prous JR. Evidence for a crucial modulating role of the sodium channel in the QTc prolongation related to antipsychotics. J Psychopharmacol. 2014, (28):329-34010.1177/026988111351506424327451Search in Google Scholar

[11] Silvestre JS, Prous JR. Comparative evaluation of hERG potassium channel blockade by antipsychotics. Methods Find Exp Clin Pharmacol. 2007, 29: 457-46510.1358/mf.2007.29.7.111917217982510Search in Google Scholar

[12] Taggart P, Sutton P, Chalabi Z, et al. Effect of adrenergic stimulation on action potential duration restitution in humans. Circulation. 2003,107:285-28910.1161/01.CIR.0000044941.13346.74Search in Google Scholar

[13] Wang J, Liu YS, Zhu WX, et al. Olanzapine-induced weight gain plays a key role in the potential cardiovascular risk: evidence from heart rate variability analysis. Scientific Reports. 2014 10.1038/srep07394426022525487560Search in Google Scholar