Association Between Acute Inflammatory Response and Infarct Size in Stemi Patients Undergoing Primary PCI

1. Frangogiannis NG. The immune system and the remodeling infarcted heart: cell biological insights and therapeutic opportunities. J Cardiovasc Pharmacol. 2014;63:185-195. doi: 10.1097/FJC.0000000000000003.10.1097/FJC.0000000000000003394916324072174Open DOISearch in Google Scholar

2. Ruparelia N, Godec J, Lee R, et al. Acute myocardial infarction activates distinct inflammation and proliferation pathways in circulating monocytes, prior to recruitment, and identified through conserved transcriptional responses in mice and humans. Eur Heart J. 2015;36:1923-1934. doi: 10.1093/ eurheartj/ehv195.10.1093/eurheartj/ehv195457117725982896Open DOISearch in Google Scholar

3. Lugrin J, Parapanov R, Rosenblatt-Velin N, et al. Cutting edge: IL-1α is a crucial danger signal triggering acute myocardial inflammation during myocardial infarction. J Immunol. 2015;194:499-503. doi: 10.4049/jimmunol.1401948.10.4049/jimmunol.1401948427819625505286Open DOISearch in Google Scholar

4. Kain V, Prabhu SD, Halade GV. Inflammation revisited: inflammation versus resolution of inflammation following myocardial infarction. Basic Res Cardiol. 2014;109:444. doi: 10.1007/s00395-014-0444-7.10.1007/s00395-014-0444-725248433Open DOISearch in Google Scholar

5. Nguyen TL, Phan JA, Hee L, et al. High-sensitivity troponin T predicts infarct scar characteristics and adverse left ventricular function by cardiac magnetic resonance imaging early after reperfused acute myocardial infarction. Am Heart J. 2015;170:715-725.e2. doi: 10.1016/j.ahj.2015.06.022.10.1016/j.ahj.2015.06.02226386795Open DOISearch in Google Scholar

6. Prabhu SD, Frangogiannis NG. The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis. Circ Res. 2016;119:91-112. doi: 10.1161/ CIRCRESAHA.116.303577.10.1161/CIRCRESAHA.116.303577492252827340270Open DOISearch in Google Scholar

7. Groot HE, Hartman MH1, Gu YL, et al. Soluble interleukin 6 receptor levels are associated with reduced myocardial reperfusion after percutaneous coronary intervention for acute myocardial infarction. Cytokine. 2015;73:207-212. doi: 10.1016/j.cyto.2015.02.004.10.1016/j.cyto.2015.02.00425792278Search in Google Scholar

8. Puhakka M, Magga J, Hietakorpi S, et al. Interleukin-6 and tumor necrosis factor alpha in relation to myocardial infarct size and collagen formation. J Card Fail. 2003;9:325-332. doi: 10.1054/jcaf.2003.38.10.1054/jcaf.2003.3813680554Open DOISearch in Google Scholar

9. Müller J, Gorressen S, Grandoch M, et al. Interleukin-6- dependent phenotypic modulation of cardiac fibroblasts after acute myocardial infarction. Basic Res Cardiol. 2014;109:440. doi: 10.1007/s00395-014-0440-y.10.1007/s00395-014-0440-y25236954Open DOISearch in Google Scholar

10. Takahashi T, Anzai T, Kaneko H, et al. Increased C-reactive protein expression exacerbates left ventricular dysfunction and remodeling after myocardial infarction. Am J Physiol Heart Circ Physiol. 2010;299:H1795-H1804. doi: 10.1152/ ajpheart.00001.2010.10.1152/ajpheart.00001.201020852043Search in Google Scholar

11. Zhang R, Zhang YY, Huang XR, et al. C-reactive protein promotes cardiac fibrosis and inflammation in angiotensin II-induced hypertensive cardiac disease. Hypertension. 2010;55:953-960. doi: 10.1161/HYPERTENSIONAHA.109.140608.10.1161/HYPERTENSIONAHA.109.14060820157054Open DOISearch in Google Scholar

12. Ribeiro DR, Ramos AM, Vieira PL, et al. High-sensitivity C-reactive protein as a predictor of cardiovascular events after ST-elevation myocardial infarction. Arq Bras Cardiol. 2014;103:69-75.10.5935/abc.20140086412676325120085Search in Google Scholar

13. Reinstadler SJ, Thiele H, Eitel I. Risk stratification by cardiac magnetic resonance imaging after ST-elevation myocardial infarction. Curr Opin Cardiol. 2015;30:681-689. doi: 10.1097/ HCO.0000000000000227.10.1097/HCO.000000000000022726398412Open DOISearch in Google Scholar

14. Sadahiro T, Kohsaka S, Okuda S, et al. MRI and serum highsensitivity C reactive protein predict long-term mortality in non-ischaemic cardiomyopathy. Open Heart. 2015;2:e000298. doi: 10.1136/openhrt-2015-000298.10.1136/openhrt-2015-000298462022926512328Open DOISearch in Google Scholar

15. Mason JC, Libby P. Cardiovascular disease in patients with chronic inflammation: mechanisms underlying premature cardiovascular events in rheumatologic conditions. Eur Heart J. 2015;36:482-489. doi:10.1093/eurheartj/ehu403.10.1093/eurheartj/ehu403434036425433021Open DOISearch in Google Scholar

16. Xu S, Song M, Xiong Y, Liu X, He Y, Qin Z. The association between periodontal disease and the risk of myocardial infarction: a pooled analysis of observational studies. BMC Cardiovascular Disorders. 2017;17:50. doi:10.1186/s12872-017- 0480-y.10.1186/s12872-017-0480-y528686228143450Open DOISearch in Google Scholar

17. Sincar CD, Ioanid N, Rudnic I, et al. The biochemical effects of non-surgical periodontal therapy in patients with and without chronic renal disease. Revista de Chimie. 2017;68:605-607.10.37358/RC.17.3.5511Search in Google Scholar

18. Hansson GK, Libby P, Tabas I. Inflammation and plaque vulnerability. Journal of Internal Medicine. 2015;278:483-493. doi:10.1111/joim.12406.10.1111/joim.12406508211126260307Open DOISearch in Google Scholar

19. Suleiman M, Khatib R, Agmon Y et al. Early inflammation and risk of long-term development of heart failure and mortality in survivors of acute myocardial infarction predictive role of C-reactive protein. J Am Coll Cardiol. 2006;47:962-968. doi: 10.1016/j.jacc.2005.10.05510.1016/j.jacc.2005.10.05516516078Open DOISearch in Google Scholar

20. Karpiński L, Płaksej R, Kosmala W, Witkowska M. Serum levels of interleukin-6, interleukin-10 and C-reactive protein in relation to left ventricular function in patients with myocardial infarction treated with primary angioplasty. Kardiol Pol. 2008;66:1279-1285.10.20452/pamw.624Search in Google Scholar

21. Ritschel VN, Seljeflot I, Arnesen H, et al. IL-6 signalling in patients with acute ST-elevation myocardial infarction. Results Immunol. 2013;4:8-13. doi: 10.1016/j.rinim.2013.11.002.10.1016/j.rinim.2013.11.002397382124707455Open DOISearch in Google Scholar

22. Roes SD, Kelle S, Kaandorp TA, et al. Comparison of myocardial infarct size assessed with contrast-enhanced magnetic resonance imaging and left ventricular function and volumes to predict mortality in patients with healed myocardial infarction. Am J Cardiol. 2007;100:930-936. doi: 10.1016/j. amjcard.2007.04.029.10.1016/j.amjcard.2007.04.02917826372Open DOISearch in Google Scholar

23. Ørn S, Manhenke C, Ueland T, et al. C-reactive protein, infarct size, microvascular obstruction, and left-ventricular remodelling following acute myocardial infarction. Eur Heart J. 2009;30:1180-1186. doi: 10.1093/eurheartj/ehp070.10.1093/eurheartj/ehp07019299430Open DOISearch in Google Scholar

24. Khan JN, McCann GP. Cardiovascular magnetic resonance imaging assessment of outcomes in acute myocardial infarction. World J Cardiol. 2017;9:109-133. doi:10.4330/wjc. v9.i2.109.10.4330/wjc.v9.i2.109532973828289525Open DOISearch in Google Scholar

25. Rajewska-Tabor J, Pyda M, Kociemba A, Janus M, Lanocha M, Siniawski A. Influence of inflammatory response on infarct size and microvascular obstruction estimated by cardiac magnetic resonance in patients with ST-elevation myocardial infarction. J Cardiovasc Magn Reson. 2015;17(Suppl 1):P160.doi:10.1186/1532-429X-17-S1-P160.10.1186/1532-429X-17-S1-160Open DOISearch in Google Scholar

26. Fang L, Moore X-L, Dart AM, Wang L-M. Systemic inflammatory response following acute myocardial infarction. J Geriatr Cardiol. 2015;12:305-312. doi:10.11909/j.issn.1671-5411.2015.03.020.Search in Google Scholar