Requiere autenticación

The Xg blood group system: no longer forgotten


Cite

Johnson NC. XG: the forgotten blood group system. Immunohematology. 2011;27:68–71. Johnson NC. XG: the forgotten blood group system . Immunohematology. 2011 ; 27 : 68 71 . 10.21307/immunohematology-2019-177 Search in Google Scholar

Veldhuisen B, van der Schoot CE, de Haas M. Blood group genotyping: from patient to high-throughput donor screening. Vox Sang 2009;97:198–206. Veldhuisen B van der Schoot CE de Haas M. Blood group genotyping: from patient to high-throughput donor screening . Vox Sang 2009 ; 97 : 198 206 . 10.1111/j.1423-0410.2009.01209.x19548962 Search in Google Scholar

Ellis NA, Tippett P, Petty A, et al. PBDX is the XG blood group gene. Nat Genet 1994;8:285–90. Ellis NA Tippett P Petty A , PBDX is the XG blood group gene . Nat Genet 1994 ; 8 : 285 90 . 10.1038/ng1194-2857533029 Search in Google Scholar

Goodfellow PN, Tippett P. A human quantitative polymorphism related to Xg blood groups. Nature 1981;289:404. Goodfellow PN Tippett P. A human quantitative polymorphism related to Xg blood groups . Nature 1981 ; 289 : 404 . 10.1038/289404a07464908 Search in Google Scholar

Goodfellow PJ, Pritchard C, Tippett P, et al. Recombination between the X and Y chromosomes: implications for the relationship between MIC2, XG and YG. Ann Hum Genet 1987;51:161–7. Goodfellow PJ Pritchard C Tippett P , Recombination between the X and Y chromosomes: implications for the relationship between MIC2, XG and YG . Ann Hum Genet 1987 ; 51 : 161 7 . 10.1111/j.1469-1809.1987.tb01058.x3502698 Search in Google Scholar

Möller M, Lee YQ, Vidovic K, et al. Disruption of a GATA1-binding motif upstream of XG/PBDX abolishes Xga expression and resolves the Xg blood group system. Blood 2018;132: 334–8. Möller M Lee YQ Vidovic K , Disruption of a GATA1-binding motif upstream of XG/PBDX abolishes Xga expression and resolves the Xg blood group system . Blood 2018 ; 132 : 334 8 . 10.1182/blood-2018-03-84254229748255 Search in Google Scholar

Yeh C-C, Chang C-J, Twu Y-C, et al. The molecular genetic background leading to the formation of the human erythroid-specific Xga/CD99 blood groups. Blood Adv 2018;2:1854–64. Yeh C-C Chang C-J Twu Y-C , The molecular genetic background leading to the formation of the human erythroid-specific Xga/CD99 blood groups . Blood Adv 2018 ; 2 : 1854 64 . 10.1182/bloodadvances.2018018879609372530061310 Search in Google Scholar

Lane WJ, Aguad M, Smeland-Wagman R, et al. A whole genome approach for discovering the genetic basis of blood group antigens: independent confirmation for P1 and Xga. Transfusion 2019;59:908–15. Lane WJ Aguad M Smeland-Wagman R , A whole genome approach for discovering the genetic basis of blood group antigens: independent confirmation for P1 and Xga . Transfusion 2019 ; 59 : 908 15 . 10.1111/trf.15089640298630592300 Search in Google Scholar

Meynet O, Scotlandi K, Pradelli E, et al. Xg expression in Ewing’s sarcoma is of prognostic value and contributes to tumor invasiveness. Cancer Res 2010;70:3730–8. Meynet O Scotlandi K Pradelli E , Xg expression in Ewing’s sarcoma is of prognostic value and contributes to tumor invasiveness . Cancer Res 2010 ; 70 : 3730 8 . 10.1158/0008-5472.CAN-09-283720388798 Search in Google Scholar

Uhlén M, Fagerberg L, Hallström BM, et al. Tissue-based map of the human proteome. Science 2015;347:1260419. Uhlén M Fagerberg L Hallström BM , Tissue-based map of the human proteome . Science 2015 ; 347 : 1260419 . 10.1126/science.126041925613900 Search in Google Scholar

Lee YQ, Storry JR, Karamatic Crew V, et al. A large deletion spanning XG and GYG2 constitutes a genetic basis of the Xgnull phenotype, underlying anti-Xga production. Transfusion 2019;59:1843–9. Lee YQ Storry JR Karamatic Crew V , A large deletion spanning XG and GYG2 constitutes a genetic basis of the Xgnull phenotype, underlying anti-Xga production . Transfusion 2019 ; 59 : 1843 9 . 10.1111/trf.1524230938838 Search in Google Scholar

Mensah MA, Hestand MS, Larmuseau MHD, et al. Pseudoautosomal region 1 length polymorphism in the human population. PLoS Genet 2014;10:e1004578. Mensah MA Hestand MS Larmuseau MHD , Pseudoautosomal region 1 length polymorphism in the human population . PLoS Genet 2014 ; 10 : e1004578 . 10.1371/journal.pgen.1004578422260925375121 Search in Google Scholar

Irgens HU, Fjeld K, Johansson BB, et al. Glycogenin-2 is dispensable for liver glycogen synthesis and glucagon-stimulated glucose release. J Clin Endocrinol Metab 2015; 100:E767–75. Irgens HU Fjeld K Johansson BB , Glycogenin-2 is dispensable for liver glycogen synthesis and glucagon-stimulated glucose release . J Clin Endocrinol Metab 2015 ; 100 : E767 75 . 10.1210/jc.2014-433725751106 Search in Google Scholar

Poriswanish N, Neumann R, Wetton JH, et al. Recombination hotspots in an extended human pseudoautosomal domain predicted from double-strand break maps and characterized by sperm-based crossover analysis. PLoS Genet 2018;14:e1007680. Poriswanish N Neumann R Wetton JH , Recombination hotspots in an extended human pseudoautosomal domain predicted from double-strand break maps and characterized by sperm-based crossover analysis . PLoS Genet 2018 ; 14 : e1007680 . 10.1371/journal.pgen.1007680619373630296256 Search in Google Scholar

Thornton N, Karamatic Crew V, Muñiz-Diaz E, et al. Four examples of anti-CD99 and discovery of the molecular bases of the rare CD99– phenotype. Vox Sang 2015;109(Suppl 1):50–1. Thornton N Karamatic Crew V Muñiz-Diaz E , Four examples of anti-CD99 and discovery of the molecular bases of the rare CD99– phenotype . Vox Sang 2015 ; 109 (Suppl 1) : 50 1 . Search in Google Scholar

Pasello M, Manara MC, Scotlandi K. CD99 at the crossroads of physiology and pathology. J Cell Commun Signal 2018;12: 55–68. Pasello M Manara MC Scotlandi K. CD99 at the crossroads of physiology and pathology . J Cell Commun Signal 2018 ; 12 : 55 68 . 10.1007/s12079-017-0445-z584220229305692 Search in Google Scholar

Bedau T, Peters F, Prox J, et al. Ectodomain shedding of CD99 within highly conserved regions is mediated by the metalloprotease meprin β and promotes transendothelial cell migration. FASEB J 2016;31:1226–37. Bedau T Peters F Prox J , Ectodomain shedding of CD99 within highly conserved regions is mediated by the metalloprotease meprin β and promotes transendothelial cell migration . FASEB J 2016 ; 31 : 1226 37 . 10.1096/fj.201601113R28003343 Search in Google Scholar

eISSN:
1930-3955
Idioma:
Inglés
Calendario de la edición:
4 veces al año
Temas de la revista:
Medicine, Clinical Medicine, Laboratory Medicine