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Review: conditions causing weak expression of Kell system antigens


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Coombs RRA, Mourant AE, Race RR. In-vivo isosensitisation of red cells in babies with haemolytic disease. Lancet 1946;i:264–6.10.1016/S0140-6736(46)91925-3Search in Google Scholar

Daniels GL, Anstee DJ, Cartron J-P, et al. Blood group terminology 1995. From the ISBT working party on terminology for red cell surface antigens. Vox Sang 1995;69:265–79.Search in Google Scholar

Marsh WL, Redman CM. The Kell blood group system: a review. Transfusion 1990;30:158–67.10.1046/j.1537-2995.1990.30290162904.xSearch in Google Scholar

Lee S, Wu X, Reid ME, Zelinski T, Redman C. Molecular basis of the Kell (K1) phenotype. Blood 1995;85:912–6.10.1182/blood.V85.4.912.bloodjournal854912Search in Google Scholar

Lee S, Wu X, Reid ME, Redman C. Molecular basis of the K:6,–7 [Js(a+b–)] phenotype in the Kell blood group system. Transfusion 1995;35:822–5.10.1046/j.1537-2995.1995.351096026362.xSearch in Google Scholar

Lee S, Wu X, Son S, et al. Point mutations characterize KEL10, the KEL3, KEL4, and KEL21 alleles, and the KEL17 and KEL11 alleles. Transfusion 1996;36:490–4.10.1046/j.1537-2995.1996.36696269505.xSearch in Google Scholar

Advani H, Zamor J, Judd WJ, Johnson CL, Marsh WL. Inactivation of Kell blood group antigens by 2- aminoethylisothiouronium bromide. Br J Haematol 1982;51:107–15.10.1111/j.1365-2141.1982.tb07295.xSearch in Google Scholar

Branch DR, Petz LD. Disulphide bonds are a requirement for Kell and Cartwright (Yta) blood group antigen integrity (abstract). Transfusion 1982;22:420.Search in Google Scholar

Allen FH Jr, Lewis SJ, Fudenberg H. Studies of anti- Kpb, a new antibody in the Kell blood group system. Vox Sang 1958;3:1–13.Search in Google Scholar

Tippett P. Expansion of the Lutheran and Kell blood group systems. In: Mohn JF, Plunkett RW, Cunningham RK, Lambert RM, eds. Human blood groups. Basel, Karger, 1977:401–9.Search in Google Scholar

Ford DS, Knight AE, Smith F. A further example of Kpa/Ko exhibiting depression of some Kell group antigens. Vox Sang 1977;32:220–3.Search in Google Scholar

Marsh WL, Jensen L, Øyen R, et al. Anti-K13 and the K:–13 phenotype. A blood-group variant related to the Kell system. Vox Sang 1974;26:34–40.Search in Google Scholar

Kaita H, Lewis M, Chown B, Gard E. A further example of the Kell blood group phenotype K–, k–, Kp(a–b–). Nature 1959;183:1586.10.1038/1831586b0Search in Google Scholar

Nunn HD, Giles DM, Dormandy KM. A second example of anti-Ku in a patient who has the rare Kell phenotype, Ko. Vox Sang 1966;11:611–9.10.1111/j.1423-0410.1966.tb04258.xSearch in Google Scholar

Marsh WL, Øyen R, Nichols ME, Allen FH Jr. Chronic granulomatous disease and the Kell blood groups. Br J Haematol 1975;29:247–62.10.1111/j.1365-2141.1975.tb01819.xSearch in Google Scholar

Marsh WL, Redman CM. Recent developments in the Kell blood group system. Transfus Med Rev 1987; 1:4–20.10.1016/S0887-7963(87)70002-9Search in Google Scholar

Peloquin P, Yochum G, Hagy L, Øyen R, Johnson C. The Mullins phenotype: another RBC phenotype characterized by weak Kell antigens (abstract). Transfusion 1988;28(Suppl):19S.Search in Google Scholar

Brown A, Berger R, Lasko D, et al. The Day phenotype: a “new” variant in the Kell blood group system. Revue Française de Transfusion et Immuno-hématologie 1982;25:619–27.10.1016/S0338-4535(82)80070-6Search in Google Scholar

Marsh WL, Schnipper EF, Johnson CL, Mueller KA, Schwartz SA. An individual with McLeod syndrome and the Kell blood group antigen K(K1). Transfusion 1983;23:336–8.10.1046/j.1537-2995.1983.23483276871.xSearch in Google Scholar

White W, Washington ED, Sabo BH, et al. Anti-Km in a transfused man with McLeod syndrome. Revue Française de Transfusion et Immuno-hématologie 1980;23:305–17.10.1016/S0338-4535(80)80134-6Search in Google Scholar

Øyen R, Reid ME, Rubinstein P, Ralph H. A method to detect McLeod phenotype red blood cells. Immunohematology 1996;12:160–3.10.21307/immunohematology-2019-771Search in Google Scholar

Arndt P, Garratty G, Raghunathan R. Use of flow cytometry for quantitation of mixed RBC populations in female carriers of the McLeod gene (abstract). Transfusion 1994;34(Suppl):21S.Search in Google Scholar

Daniels GL, Weinauer F, Stone C, et al. A combination of the effects of rare genotypes at the XK and KEL blood group loci results in absence of Kell system antigens from the red blood cells. Blood 1996;88:4045–50.10.1182/blood.V88.10.4045.bloodjournal88104045Search in Google Scholar

Race RR, Sanger R. Blood groups in man. Oxford, England: Blackwell Scientific Publications, 1975.Search in Google Scholar

Reid ME. The Gerbich blood group antigens: a review. Med Lab Sci 1986;43:177–82.Search in Google Scholar

Daniels GL. Studies on Gerbich negative phenotypes and Gerbich antibodies (abstract). Transfusion 1982;22:405.Search in Google Scholar

Anstee DJ, Ridgwell K, Tanner MJ, Daniels GL, Parsons SF. Individuals lacking the Gerbich blood- group antigen have alterations in the human erythrocyte membrane sialoglycoproteins beta and gamma. Biochem J 1984;221:97–104.10.1042/bj221009711440076466322Search in Google Scholar

Reid ME, Anstee DJ, Tanner MJ, Ridgwell K, Nurse GT. Structural relationships between human erythrocyte sialoglycoproteins beta and gamma and abnormal sialoglycoproteins found in certain rare human erythrocyte variants lacking the Gerbich blood-group antigen(s). Biochem J 1987;244:123–8.10.1042/bj244012311479622444210Search in Google Scholar

Reid ME. Biochemistry and molecular cloning analysis of human red cell sialoglycoproteins that carry Gerbich blood group antigens. In: Unger P, Laird-Fryer B, eds. Blood group systems: MNSsU, Gerbich and glycoproteins. Arlington, VA: American Association of Blood Banks, 1989:73–103.Search in Google Scholar

Anstee DJ. Blood group-active surface molecules of the human red blood cell. Vox Sang 1990;58:1–20.10.1111/j.1423-0410.1990.tb02049.x2180209Search in Google Scholar

Seyfreid H, Górska B, Maj S, Sylwestrowicz T, Giles CM, Goldsmith KLG. Apparent depression of antigens of the Kell blood group system associated with autoimmune acquired haemolytic anaemia. Vox Sang 1972;23:528–36.10.1111/j.1423-0410.1972.tb03846.x4657687Search in Google Scholar

Marsh WL, Øyen R, Alicea E, Linter M, Horton S. Autoimmune hemolytic anemia and the Kell blood groups. Am J Hematol 1979;7:155–62.10.1002/ajh.2830070208539592Search in Google Scholar

Beck ML, Marsh WL, Pierce SR, DiNapoli J, Øyen R, Nichols ME. Auto anti-Kpb associated with weakened antigenicity in the Kell blood group system: a second example. Transfusion 1979;19:197–202.10.1046/j.1537-2995.1979.19279160294.x86215Search in Google Scholar

Williamson LM, Poole J, Redman C, et al. Transient loss of proteins carrying Kell and Lutheran red cell antigens during constructive relapses of autoimmune thrombocytopenia. Br J Haematol 1994;87:805–12.10.1111/j.1365-2141.1994.tb06741.x7527243Search in Google Scholar

Marsh WL, DiNapoli J, Øyen R. Auto-immune hemolytic anemia caused by anti-K13. Vox Sang 1979;36:174–8.10.1111/j.1423-0410.1979.tb04419.x462904Search in Google Scholar

Manny N, Levene C, Sela R, Johnson CL, Mueller KA, Marsh WL. Autoimmunity and the Kell blood groups: auto-anti-Kpb in a Kp(a+b–) patient. Vox Sang 1983;45:252–6.Search in Google Scholar

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Medicine, Clinical Medicine, Laboratory Medicine