Zitieren

1. Reinke JM, Sorg H. Wound repair and regeneration. Eur Surg Res. 2012 Jul 11;49(1):35–43.10.1159/00033961322797712 Search in Google Scholar

2. 21. Gurtner GC, Werner S, Barrandon Y, Longaker MT. Wound repair and regeneration. Nature. 2008 May 15;453(7193):314–21.10.1038/nature0703918480812 Search in Google Scholar

3. Sinno H, Prakash S. Complements and the wound healing cascade: an updated review. Plast Surg Int. 2013 Jul 24;2013:146764.10.1155/2013/146764374199323984063 Search in Google Scholar

4. Pandolfi F, et al. Key role of DAMP in inflammation, cancer, and tissue repair. Clin Ther. 2016;38(5):1017–2810.1016/j.clinthera.2016.02.02827021609 Search in Google Scholar

5. Venereau E, Ceriotti C, Bianchi ME. DAMPs from Cell Death to New Life. Front Immunol. 2015;6:422.10.3389/fimmu.2015.00422453955426347745 Search in Google Scholar

6. Muller, S. et al. The double life of HMGB1 chromatin protein: architectural factor and extracellular signal. EMBO J. 16, 4337–4340 (2001). Search in Google Scholar

7. Scaffidi P, Misteli T, Bianchi ME. Release of chromatin protein HMGB1 by necrotic cells triggers inflammation. Nature. 2002;418(6894):191–5.10.1038/nature0085812110890 Search in Google Scholar

8. Ranzato E, Martinotti S, Pedrazzi M, Patrone M. High mobility group box protein-1 in wound repair. Cells. 2012;1(4):699–710. Published 2012 Sep 28.doi:10.3390/ cells104069910.3390/cells1040699390115324710526 Search in Google Scholar

9. Straino et al. High-mobility group box 1 protein in human and murine skin: involvement in wound healing. J Invest Dermatol. 2008 Jun;128(6):1545–53. doi: 10.1038/sj.jid.5701212. Epub 2008 Jan 3110.1038/sj.jid.570121218239618 Search in Google Scholar

10. C, Bustin M, Talwar S, Tropea M, Gerstenberger E, Shelhamer JH et al. (2003) Inflammation-promoting activity of HMGB1 on human microvascular endothelial cells. Blood 101:2652–6010.1182/blood-2002-05-130012456506 Search in Google Scholar

11. Huang JM, Hu J, Chen N, Hu ML. Relationship between plasma high-mobility group box1 levels and clinical outcomes of ischemic stroke. Journal of critical care. 2013c; 28(5):792–797.10.1016/j.jcrc.2012.10.00323137435 Search in Google Scholar

12. Yao Y, Su Z, Yang Y, Kao R, Martin CM, Rui T. Endogenous HMGB1 contributes to ischemia-reperfusion-induced myocardial apoptosis by potentiating the effect of TNF-α/JNK. Am J Physiol Heart Circ Physiol. 2011b; 300(3):H913–H921.10.1152/ajpheart.00703.2010330219421186276 Search in Google Scholar

13. Peltz ED, Moore EE, Eckels PC, Damle SS, Tsuruta Y, Johnson JL, Sauaia A, Silliman CC, Banerjee A, Abraham E. HMGB1 is markedly elevated within 6 hours of mechanical trauma in humans. Shock (Augusta, Ga.). 2009; 32(1):17–22. Search in Google Scholar

14. A. Bierhaus, P.M. Humpert, M. Morcos, T. Wendt, T. Chavakis, B. Arnold, D.M. Stern, P.P. Nawroth, Understanding RAGE, the receptor for advanced glycation end products, J. Mol. Med. 83 (2005) 876–88610.1007/s00109-005-0688-716133426 Search in Google Scholar

15. G.P. Sims, D.C. Rowe, S.T. Rietdijk, R. Herbst, A.J. Coyle, HMGB1 and RAGE in inflammation and cancer, Annu. Rev. Immunol. 28 (2010) 367–388.10.1146/annurev.immunol.021908.13260320192808 Search in Google Scholar

16. Sorci G, Riuzzi F, Giambanco I, Donato R. RAGE in tissue homeostasis, repair and regeneration. biochim biophys Acta. 2013 Jan;1833(1):101–9.10.1016/j.bbamcr.2012.10.02123103427 Search in Google Scholar

17. Kim HJ, Jeong MS, Jang SB. Molecular Characteristics of RAGE and Advances in Small-Molecule Inhibitors. Int J Mol Sci. 2021 Jun 27;22(13):6904.10.3390/ijms22136904826810134199060 Search in Google Scholar

18. Schmidt AM, Yan SD, Yan SF, Stern DM (2001) The multiligand receptor RAGE is a progression factor amplifying immune and inflammatory responses J Clin Invest 108:949–95510.1172/JCI200114002 Search in Google Scholar

19. Szczepanski MJ, Luczak M, Olszewska E, Molinska-Glura M, Zagor M, Krzeski A, Skarzynski H, Misiak J, Dzaman K, Bilusiak M, Kopec T, Leszczynska M, Witmanowski H, Whiteside TL. Molecular signaling of the HMGB1/RAGE axis contributes to cholesteatoma pathogenesis. J Mol Med (Berl). 2015 Mar;93(3):305–14.10.1007/s00109-014-1217-3433330125385222 Search in Google Scholar

20. Ramasamy R, Vannucci SJ, Yan SS, Herold K, Yan SF, Schmidt AM. Advanced glycation end products and RAGE: a common thread in aging, diabetes, neurodegeneration, and inflammation.Glycobiology. 2005 Jul;15(7):16R–28R.10.1093/glycob/cwi05315764591 Search in Google Scholar

21. Ji XY, Chen Y, Ye GH, Dong MW, Lin KZ, Han JG, Feng XP, Li XB, Yu LS, Fan YY. Detection of RAGE expression and its application to diabetic wound age estimation. Int J Legal Med. 2017 May;131(3):691–698.10.1007/s00414-016-1529-728078446 Search in Google Scholar

22. Sparvero LJ, Asafu-Adjei D, Kang R, Tang D, Amin N, Im J, Rutledge R, Lin B, Amoscato AA, Zeh HJ, Lotze MT. RAGE (Receptor for Advanced Glycation Endproducts), RAGE ligands, and their role in cancer and inflammation. J Transl Med. 2009 Mar 17;7:17.10.1186/1479-5876-7-17266664219292913 Search in Google Scholar

23. Rauvala H, Rouhiainen A (2007) RAGE as a receptor of HMGB1 (Amphoterin): roles in health and disease. Curr Mol Med 7:725–73410.2174/15665240778322075018331230 Search in Google Scholar

24. Kikuchi K, Kawahara KI, Biswas KK, et al. HMGB1: A new marker for estimation of the postmortem interval. Exp Ther Med. 2010;1(1):109–111.10.3892/etm_00000019349037923136602 Search in Google Scholar

25. Ahmed Alaa El-Din E, Mohamed Ahmed S, Abdallah El Shafei D, El-Sayed Mostafa H. Implication of High-mobility group box-1 and skin post mortem changes in estimation of time passed since death: Animal and human study. Leg Med (Tokyo). 2021 Nov;53:10.1016/j.legalmed.2021.10194934333193 Search in Google Scholar

26. Gao TL, Yuan XT, Yang D, Dai HL, Wang WJ, Peng X, Shao HJ, Jin ZF, Fu ZJ. Expression of HMGB1 and RAGE in rat and human brains after traumatic brain injury. J Trauma Acute Care Surg. 2012 Mar;72(3):643–9.10.1097/TA.0b013e31823c54a622491548 Search in Google Scholar

27. Li J, Qu X, Schmidt AM. Sp1-binding elements in the promoter of RAGE are essential for amphoterin-mediated gene expression in cultured neuroblastoma cells. J Biol Chem. 1998 Nov 20;273(47):30870–810.1074/jbc.273.47.308709812979 Search in Google Scholar

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