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An update on the Lewis blood group system

   | 15. Feb. 2020

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Combs MR. Lewis blood group system review. Immunohematology 2009;25:112–8.CombsMR.Lewis blood group system review. Immunohematology2009;25:1128.10.21307/immunohematology-2019-243Search in Google Scholar

Ewald D, Sumner S. Blood type biochemistry and human disease. Wiley Interdiscip Rev Syst Biol Med 2016;8:517–35.EwaldDSumnerS.Blood type biochemistry and human disease. Wiley Interdiscip Rev Syst Biol Med2016;8:51735.10.1002/wsbm.1355506161127599872Search in Google Scholar

Arifuzzaman M, Ahmed T, Rahman MA, et al. Individuals with Le(a+b–) blood group have increased susceptibility to symptomatic Vibrio cholera O1 infection. PLoS NTDS 2011;5:e1413.ArifuzzamanMAhmedTRahmanMA, Individuals with Le(a+b–) blood group have increased susceptibility to symptomatic Vibrio cholera O1 infection. PLoS NTDS2011;5:e1413.10.1371/journal.pntd.0001413324645122216364Search in Google Scholar

Blackwell CC, Jonsdottir K, Weir DM, et al. Blood group, secretor status and susceptibility to bacterial meningitis. FEMS Microbiol Immunol 1989;1:351–6.BlackwellCCJonsdottirKWeirDM, Blood group, secretor status and susceptibility to bacterial meningitis. FEMS Microbiol Immunol1989;1:3516.10.1111/j.1574-6968.1989.tb02420.x2698730Search in Google Scholar

Greenwell P. Blood group antigens: molecules seeking a function? Glycoconj J 1997;14:159–73.GreenwellP.Blood group antigens: molecules seeking a function?Glycoconj J1997;14:15973.10.1023/A:1018581503164Search in Google Scholar

Smyth DJ, Cooper JD, Howson JM, et al. FUT2 nonsecretor status links type 1 diabetes susceptibility and resistance to infection. Diabetes 2011;60:3081–4.SmythDJCooperJDHowsonJM, FUT2 nonsecretor status links type 1 diabetes susceptibility and resistance to infection. Diabetes2011;60:30814.10.2337/db11-0638319805722025780Search in Google Scholar

Cooling L. Blood groups in infection and host susceptibility. Clin Microbiol Rev 2015;28:801–70.CoolingL.Blood groups in infection and host susceptibility. Clin Microbiol Rev2015;28:80170.10.1128/CMR.00109-14447564426085552Search in Google Scholar

Nanthakumar NN, Dai D, Newburg DS, Walker WA. The role of indigenous microflora in the development of murine intestinal fucosyl- and sialyltransferases. FASEB J 2003;17:44–6.NanthakumarNNDaiDNewburgDSWalkerWA.The role of indigenous microflora in the development of murine intestinal fucosyl- and sialyltransferases. FASEB J2003;17:446.10.1096/fj.02-0031fje12475916Search in Google Scholar

Höglund P, Ragnhild R-L, Wikman AT. A severe haemolytic transfusion reaction caused by anti-Lea active at 37°C. Blood Transfus 2013;11:456–9.HöglundPRagnhildR-LWikmanAT.A severe haemolytic transfusion reaction caused by anti-Lea active at 37°C. Blood Transfus2013;11:4569.Search in Google Scholar

Duncan V, Pham H, Williams L III. A possible case of a haemolytic transfusion reaction caused by anti-Lea antibody. Blood Transfus 2015;13:535–6.DuncanVPhamHWilliamsLIII.A possible case of a haemolytic transfusion reaction caused by anti-Lea antibody. Blood Transfus2015;13:5356.Search in Google Scholar

Irani MS, Figueroa D, Savage G. Acute hemolytic transfusion reaction due to anti-Leb. Transfusion 2015;55:2486–8.IraniMSFigueroaDSavageG.Acute hemolytic transfusion reaction due to anti-Leb. Transfusion2015;55:24868.10.1111/trf.1317826018602Search in Google Scholar

eISSN:
1930-3955
Sprache:
Englisch
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Fachgebiete der Zeitschrift:
Medizin, Klinische Medizin, Laboratoriumsmedizin