Blood transfusion in hemolytic anemias

[1]Crosby WH, Rappaport H. Reticulocytopenia in autoimmune hemolytic anemia. Blood 1956; 11:929-36.10.1182/blood.V11.10.929.929Search in Google Scholar

[2]Conley CL, Lippman SM, Ness PM, Petz LD, Branch DR, Gallagher MT. Autoimmune hemolytic anemia with reticulocytopenia and erythroid marrow. N Engl ĵ Med 1982; 306:281-6.10.1056/NEJM198202043060507Search in Google Scholar

[3]Garratty G, Petz LD. Transfusing patients with autoimmune haemolytic anaemia. Lancet 1993; 341:1220.10.1016/0140-6736(93)91056-RSearch in Google Scholar

[4]Petz LD, Garratty G. Acquired immune hemolytic anemias. New York: Churchill Livingstone, 1980.Search in Google Scholar

[5]Branch DR, Petz LD. A new reagent ZZAP having multiple applications in immunohematology. Am J of Clin Pathol 1982;78:161-7.10.1093/ajcp/78.2.161Search in Google Scholar

[6]Vengelen-Tyler V ed. Technical manual. 12th ed. Bethesda, MD: American Association of Blood Banks, 1996.Search in Google Scholar

[7]Petz LD. Autoimmune hemolytic anemia. Hum Pathol 1983; 14:251-5.10.1016/S0046-8177(83)80026-4Search in Google Scholar

[8]Petz LD. Blood transfusion in acquired hemolytic anemia. In: Petz LD, Swisher SN, Kleinman S, Spence RK, Strauss RG, eds. Clinical practice of transfusion medicine. 3rd ed. New York: Churchill Livingstone, 1996:469-99.Search in Google Scholar

[9]9· MenitoveJE,ed. Standards for blood banks and transfusion services. 18th ed. Bethesda, MD: American Association of Blood Banks, 1997.Search in Google Scholar

[10]Jefferies LC. Transfusion therapy in autoimmune hemolytic anemia. Hematology/Oncology Clinics of North America 1994;8:1087-1104.10.1016/S0889-8588(18)30123-0Search in Google Scholar

[11]Morel PA, Bergen MO, Frank BA. A simple method for the detection of alloantibody in the presence of warm autoantibody (abstract). Transfusion 1978; 18:388.Search in Google Scholar

[12]Wa?lhermfechtel MA, Pohl BA, Chaplin H. Alloimmunization in patients with warm autoanti- bodies. A retrospective study employing three donor alloabsorptions to aid in antibody detection. Transfusion 1984;24:482-5.10.1046/j.1537-2995.1984.24685066805.x6506178Search in Google Scholar

[13]13· Laine ML, Beattie KM. Frequency of alloantibodies accompanying autoantibodies. Transfusion 1985; 25:545-6.10.1046/j.1537-2995.1985.25686071427.x4071599Search in Google Scholar

[14]James P, Rowe GP, Tozzo GG. Elucidation of alloantibodies in autoimmune haemolytic anaemia. Vox Sang 1988;54:167-71.10.1111/j.1423-0410.1988.tb03893.x3369137Search in Google Scholar

[15]Sokol RJ, Hewitt S, Booker DJ, Morris BM. Patients with red cell autoantibodies: selection of blood for transfusion. Clin Lab Haematol 1988;10:257-64.10.1111/j.1365-2257.1988.tb00019.x3180694Search in Google Scholar

[16]Issitt PD, Combs MR, Bumgarner DJ, Alien J, Kirkland A, Melroy-Carawan H. Studies of antibodies in the sera of patients who have made red cell autoantibodies. Transfusion 1996;36:481-6.10.1046/j.1537-2995.1996.36696269503.xSearch in Google Scholar

[17]Leger RM, Garratty G. Evaluation of methods for detecting alloantibodies underlying warm autoantibodies. Transfusion 1999; 39:11-6.10.1046/j.1537-2995.1999.39199116889.xSearch in Google Scholar

[18]Rosenfield RE, Jagathambal. Transfusion therapy for autoimmune hemolytic anemia. Seminars Hematol 1976; 13:311-21.Search in Google Scholar

[19]Chaplin H. Special problems in transfusion management of patients with autoimmune hemolytic anemia. In: Bell CA, ed. A seminar on laboratory management of hemolysis. Washington, D.C.: American Association of Blood Banks, 1979:135-60.Search in Google Scholar

[20]Bilgrami S, Cable R, Pisciotto P. Rowland F, Greenberg B. Fatal disseminated intravascular coagulation and pulmonary thrombosis following blood transfusion in a patient with severe autoimmune hemolytic anemia and human immunodeficiency virus infection. Transfusion 1994;34:248-52.10.1046/j.1537-2995.1994.34394196624.xSearch in Google Scholar

[21]Salama A, Bergheofer H, Mueller-Eckhardt C. Red blood cell transfusion in warm-type autoimmune haemolytic anaemia. Lancet 1992;340:1515-7.10.1016/0140-6736(92)92766-9Search in Google Scholar

[22]Petz LD, Calhoun L, Shulman IA, Johnson C, Herron RM. The sickle cell hemolytic transfusion reaction syndrome. Transfusion 1997;37:382-92.10.1046/j.1537-2995.1997.37497265338.x9111275Search in Google Scholar

[23]Garratty G. Severe reactions associated with transfusion of patients with sickle cell disease (editorial). Transfusion 1997;37:357-61.10.1046/j.1537-2995.1997.37497265334.x9111271Search in Google Scholar

[24]Sirchia G, Morelati F, Rebulla P The sickle cell hemolytic transfusion reaction syndrome (letter). Transfusion 1997; 37:1098-9.10.1046/j.1537-2995.1997.371098016453.x9354833Search in Google Scholar

[25]Petz LD. The expanding boundaries of transfusion medicine. In: Nance SJ, ed. Clinical and basic science aspects of immunohematology. Arlington, VA: American Association of Blood Banks, 1991:73-113.Search in Google Scholar

[26]Chaplin HJ, Zarkowsky HS. Combined sickle cell disease and autoimmune hemolytic anemia. Arch Intern Med 1981; 141.1091-3.10.1001/archinte.141.8.1091Search in Google Scholar

[27]Sosler SD, Perkins JT, Saporito C, et al. Severe autoimmune hemolytic anemia induced by transfusion in two alloimmunized patients with sickle cell disease (abstract). Transfusion 1989;29:498.Search in Google Scholar

[28]Thompson RA, Rowe DS. Reactive haemolysis—a distinctive form of red cell lysis. Immunology 1968;14:745-62.Search in Google Scholar

[29]Thompson RA, Lachmann PJ. Reactive lysis: the complement-mediated lysis of unsensitized cells. I. The characterization of the indicator factor and its identification as C7. J Exp Med 1970;131:629-41.10.1084/jem.131.4.62921387834193934Search in Google Scholar

[30]lachmann PJ,Thompson RA. Reactive lysis: the complement-mediated lysis of unsensitized ceils. II. The characterization of activated reactor as C5b and the participation of C8 and C9. J Exp Med 1970; 131:643-57.10.1084/jem.131.4.64321387704193935Search in Google Scholar

[31]Yachnin S, Ruthenberg JM. The initiation and enhancement of human red cell lysis by activators of the first component of complement and by first component esterase: studies using normal red cells and red cells from patients with paroxysmal nocturnal hemoglobinuria. J Clin Invest 1965;44:518-34.10.1172/JCI10516529251914278168Search in Google Scholar

[32]Sirchia G,Ferrone S,Mercuriali E Leukocyte antigenantibody reaction and lysis of paroxysmal nocturnal hemoglobinuria erythrocytes. Blood 1970; 36:334-6.10.1182/blood.V36.3.334.334Search in Google Scholar

[33]Salama A, Mueller-Eckhardt C. Delayed hemolytic transfusion reactions. Evidence for complement activation involving allogeneic and autologous red cells. Transfusion 1984;24:188-93.10.1046/j.1537-2995.1984.24384225018.x6610232Search in Google Scholar

[34]Ness PM, Shirey RS,Thoman SK, Buck SA. The differentiation of delayed serologic and delayed hemolytic transfusion reactions: incidence, long-term serologic findings, and clinical significance. Transfusion 1990;30:688-93.10.1046/j.1537-2995.1990.30891020325.x2219254Search in Google Scholar

[35]Test ST, Woolworth VS. Defective regulation of complement by the sickle erythrocyte: evidence for a defect in control of membrane attack complex formation. Blood 1994;83:842-52.10.1182/blood.V83.3.842.842Search in Google Scholar

[36]Gajewski JL, Petz LD, Calhoun L, et al. Hemolysis of transfused group O red blood cells in minor ABO- incompatible unrelated-donor bone marrow transplants in patients receiving cyclosporine without posttransplant methotrexate. Blood l·992;79: 3076-85.10.1182/blood.V79.11.3076.bloodjournal79113076Search in Google Scholar

[37]Hows J, Beddow K, Gordon-Smith E, Branch DR, Sprüce W, Sniecinski I, et al. Donor-derived red blood cell antibodies and immune hemolysis after allogeneic bone marrow transplantation. Blood 1986;67:177-81.10.1182/blood.V67.1.177.177Search in Google Scholar

[38]Greeno E, Ilstrup S, Perry E, Weidorf DJ. Total erythrocyte hemolysis following ABO minor incompatible bone marrow transplantation: rapid appearance of donor-derived red cell antibodies (abstract). Blood 1992;80 (Suppl l):484a.Search in Google Scholar

[39]Haas RJ, Rieber P, Helmig M, Strobel E, Belohradsky BH, Heim MU. Acquired immune haemolysis by anti- Aļ antibody following bone marrow transplantation. Blut 1986;53:401-4.10.1007/BF003211023535932Search in Google Scholar

[40]Herzog ft Korinkova R Stambergova M, Lukasova M. Auto anti-Aj and auto anti-NAl after bone marrow transplantation. Folia Haematol īnt Mag Klin Morphol Blutibrsch 1987;114:874-80.Search in Google Scholar

[41]Oziel-Taieb S, Faucher-Barbey C, Chabannon C, et al. Early and fatal immune haemolysis after so-called ‘ minor’ABO-incompatible peripheral blood stem cell allotransplantation. Bone Marrow Transplantation 1997;19:1155-6.10.1038/sj.bmt.17007949193761Search in Google Scholar

[42]Toren A, Dacosta Y, Manny N, et al. Passenger B-lym- phocyte-induced severe hemolytic disease after allogeneic peripheral blood stem cell transplantation. Blood 1996;87:843-4.10.1182/blood.V87.2.843.bloodjournal872843Search in Google Scholar

[43]Laurencet FM, Samii K, Bressoud A, et al. Massive delayed hemolysis following peripheral blood stem cell transplantation with minor ABO incompatibility. Hematol CellTher 1997;39:159-62.10.1007/s00282-997-0159-4Search in Google Scholar

[44]Donegan CC, Macllwaine WA, Leavell BS. Hematologic studies on patients with sickle cell anemia following multiple transfusions. Am J Med 1954;17:29.10.1016/0002-9343(54)90140-0Search in Google Scholar

[45]King KE, Shirey RS, Lankiewicz MW,Young-Ramsaran J, Ness PM. Delayed hemolytic transfusion reactions in sickle cell disease: simultaneous destruction of recipients’red cells. Transfusion 1997;37:376-81.10.1046/j.1537-2995.1997.37497265337.x9111274Search in Google Scholar