[
Alexiadis M, Eriksson N, Jamieson S, Davis M, Drummond AE, Chu S, Clyne CD, Muscat GE, Fuller PJ. Nuclear receptor profiling of ovarian granulosa cell tumors. Horm Cancer 2, 157–169, 2011.
]Search in Google Scholar
[
Beard JA, Tenga A, Chen T. The interplay of NR4A receptors and the oncogene-tumor suppressor networks in cancer. Cell Signal 27, 257–266, 2015.
]Search in Google Scholar
[
Bujnakova Mlynarcikova A, Scsukova S. Effect of selected bisphenol derivatives on nuclear receptor expression in ovarian cell line COV434. Endocr Regul 54, 275–283, 2020.
]Search in Google Scholar
[
Cao LY, Ren XM, Li CH, Zhang J, Qin WP, Yang Y, Wan B, Guo LH. Bisphenol AF and Bisphenol B exert higher estrogenic effects than Bisphenol A via G protein-coupled estrogen receptor pathway. Environ Sci Technol 51, 11423–11430, 2017.
]Search in Google Scholar
[
Cao Y, Qu X, Ming Z, Yao Y, Zhang Y. The correlation between exposure to BPA and the decrease of the ovarian reserve. Int J Clin Exp Pathol 11, 3375–3382, 2018.
]Search in Google Scholar
[
Castellini C, Totaro M, Parisi A, D’Andrea S, Lucente L, Cordeschi G, Francavilla S, Francavilla F, Barbonetti A. Bisphenol A and male fertility: Myths and realities. Front Endocrinol (Lausanne) 11, 353, 2020.
]Search in Google Scholar
[
Castillo Sanchez R, Gomez R, Perez Salazar E. Bisphenol A induces migration through a GPER-, FAK-, Src-, and ERK2-dependent pathway in MDA-MB-231 breast cancer cells. Chem Res Toxicol 29, 285–295, 2016.
]Search in Google Scholar
[
Chen D, Kannan K, Tan H, Zheng Z, Feng YL, Wu Y, Widelka M. Bisphenol analogues other than BPA: environmental occurrence, human exposure, and toxicity-A review. Environ Sci Technol 50, 5438–5453, 2016.
]Search in Google Scholar
[
Chen F, Kolben T, Meister S, Czogalla B, Kolben TM, Hester A, Burges A, Trillsch F, Schmoeckel E, Mayr D, Mayer-hofer A, Mahner S, Jeschke U, Beyer S. The role of resveratrol, Sirtuin1 and RXRα as prognostic markers in ovarian cancer. Arch Gynecol Obstet 305, 1559–1572, 2022.
]Search in Google Scholar
[
Daikoku T, Tranguch S, Chakrabarty A, Wang D, Khabele D, Orsulic S, Morrow JD, Dubois RN, Dey SK. Extracellular signal-regulated kinase is a target of cyclooxygenase-1-peroxisome proliferator-activated receptor-delta signaling in epithelial ovarian cancer. Cancer Res 67, 5285–5292, 2007.
]Search in Google Scholar
[
De Sousa Damiao R, Fujiyama Oshima CT, Stavale JN, Goncalves WJ. Analysis of the expression of estrogen receptor, progesterone receptor and chicken ovalbumin upstream promoter-transcription factor I in ovarian epithelial cancers and normal ovaries. Oncol Rep 18, 25–32, 2007.
]Search in Google Scholar
[
Delgado E, Boisen MM, Laskey R, Chen R, Song C, Sallit J, Yochum ZA, Andersen CL, Sikora MJ, Wagner J, Safe S, Elishaev E, Lee A, Edwards RP, Haluska P, Tseng G, Schurdak M, Oesterreich S. High expression of orphan nuclear receptor NR4A1 in a subset of ovarian tumors with worse outcome. Gynecol Oncol 41, 348–356, 2016.
]Search in Google Scholar
[
Ding W, Zhang Y, Cai H, Liu G, Ye Y, Xu G, Wang H, Xiong D, Zhang C, Huang Z, Luo Q. Overexpression of COUP-TFII suppresses proliferation and metastasis of human gastric cancer cells. Mol Med Rep 17, 2393–2401, 2018.
]Search in Google Scholar
[
Gerona RR, Pan J, Zota AR, Schwartz JM, Friesen M, Taylor JA, Hunt PA, Woodruff TJ. Direct measurement of Bisphenol A (BPA), BPA glucuronide and BPA sulfate in a diverse and low-income population of pregnant women reveals high exposure, with potential implications for previous exposure estimates: a cross-sectional study. Environ Health 15, 50, 2016.
]Search in Google Scholar
[
Grimaldi M, Boulahtouf A, Toporova L, Balaguer P. Functional profiling of bisphenols for nuclear receptors. Toxicology 420, 39–45, 2019.
]Search in Google Scholar
[
Hawkins SM, Loomans HA, Wan YW, Ghosh-Choudhury T, Coffey D, Xiao W, Liu Z, Sangi-Haghpeykar H, Anderson ML. Expression and functional pathway analysis of nuclear receptor NR2F2 in ovarian cancer. J Clin Endocrinol Metab 98, E1152–E1162, 2013.
]Search in Google Scholar
[
Hui L, Li H, Lu G, Chen Z, Sun W, Shi Y, Fu Z, Huang B, Zhu X, Lu W, Xia D, Wu Y. Low dose of Bisphenol A modulates ovarian cancer gene expression profile and promotes epithelial to mesenchymal transition via canonical Wnt pathway. Toxicol Sci 164, 527–538, 2018.
]Search in Google Scholar
[
Kaiser PC, Korner M, Kappeler A, Aebi S. Retinoid receptors in ovarian cancer: expression and prognosis. Ann Oncol 16, 1477–1487, 2005.
]Search in Google Scholar
[
Langdon SP, Herrington CS, Hollis RL, Gourley C. Estrogen signaling and its potential as a target for therapy in ovarian cancer. Cancers (Basel) 12, 1647, 2020.
]Search in Google Scholar
[
Lathi RB, Liebert CA, Brookfield KF, Taylor JA, vom Saal FS, Fujimoto VY, Baker VL. Conjugated bisphenol A in maternal serum in relation to miscarriage risk. Fertil Steril 102, 123–128, 2014.
]Search in Google Scholar
[
Li CH, Zhang DH, Jiang LD, Qi Y, Guo LH. Binding and activity of bisphenol analogues to human peroxisome proliferator-activated receptor β/δ. Ecotoxicol Environ Saf 226, 112849, 2021.
]Search in Google Scholar
[
Liu J, Zhang L, Lu G, Jiang R, Yan Z, Li Y. Occurrence, toxicity and ecological risk of Bisphenol A analogues in aquatic environment – A review. Ecotoxicol Environ Saf 208, 111481, 2021.
]Search in Google Scholar
[
Livak KJ, Schmittgen TC. Analysis of relative gene expression data using real-time quantitative PCR and the 2(–Delta Delta C(T)) Method. Methods 25, 402–408, 2001.
]Search in Google Scholar
[
Meinsohn MC, Smith OE, Bertolin K, Murphy BD. The orphan nuclear receptors steroidogenic factor-1 and liver receptor homolog-1: structure, regulation, and essential roles in mammalian reproduction. Physiol Rev 99, 1249–1279, 2019.
]Search in Google Scholar
[
Nishizawa H, Morita M, Sugimoto M, Imanishi S, Manabe N. Effects of in utero exposure to bisphenol A on mRNA expression of arylhydrocarbon and retinoid receptors in murine embryos. J Reprod Dev 51, 315–324, 2005.
]Search in Google Scholar
[
Nomiri S, Hoshyar R, Ambrosino C, Tyler CR, Mansouri B. A minireview of bisphenol A (BPA) effects on cancer-related cellular signaling pathways. Environ Sci Pollut Res Int 26, 8459–8467, 2019.
]Search in Google Scholar
[
Peretz J, Vrooman L, Ricke WA, Hunt PA, Ehrlich S, Hauser R, Padmanabhan V, Taylor HS, Swan SH, VandeVoort CA, Flaws JA. Bisphenol a and reproductive health: update of experimental and human evidence, 2007–2013. Environ Health Perspect 122, 775–786, 2014.
]Search in Google Scholar
[
Qi L, Guo N, Wei Q, Jin P, Wang W, Mao D. The involvement of NR4A1 and NR4A2 in the regulation of the luteal function in rats. Acta Histochem 120, 713–719, 2018.
]Search in Google Scholar
[
Rae MT, Niven D, Ross A, Forster T, Lathe R, Critchley HO, Ghazal P, Hillier SG. Steroid signalling in human ovarian surface epithelial cells: the response to interleukin-1alpha determined by microarray analysis. J Endocrinol 83, 19–28, 2004.
]Search in Google Scholar
[
Rochester J, Bolden A. Bisphenol S and F: A systematic review and comparison of the hormonal activity of Bisphenol A substitutes. Environ Health Perspect 123, 643–650, 2015.
]Search in Google Scholar
[
Sahoo PK, Aparna S, Naik PK, Singh SB, Das SK. Bisphenol A exposure induces neurobehavioral deficits and neuro-degeneration through induction of oxidative stress and activated caspase-3 expression in zebrafish brain. J Biochem Mol Toxicol 35, e22873, 2021.
]Search in Google Scholar
[
Sauer SJ, Tarpley M, Shah I, Save AV, Lyerly HK, Patierno SR, Williams KP, Devi GR. Bisphenol A activates EGFR and ERK promoting proliferation, tumor spheroid formation and resistance to EGFR pathway inhibition in estrogen receptor-negative inflammatory breast cancer cells. Carcinogenesis 38, 252–260, 2017.
]Search in Google Scholar
[
Scsukova S, Rollerova E, Bujnakova Mlynarcikova A. Impact of endocrine disrupting chemicals on onset and development of female reproductive disorders and hormone-related cancer. Reprod Biol 16, 243–254, 2016.
]Search in Google Scholar
[
Sharma S, Ahmad S, Khan MF, Parvez S, Raisuddin S. In silico molecular interaction of bisphenol analogues with human nuclear receptors reveals their stronger affinity vs. classical bisphenol A. Toxicol Mech Methods 28, 660–669, 2018.
]Search in Google Scholar
[
Toporova L, Balaguer P. Nuclear receptors are the major targets of endocrine disrupting chemicals. Mol Cell Endocrinol 502, 110665, 2020.
]Search in Google Scholar
[
Wagner N, Wagner KD. Peroxisome proliferator-activated receptors and the hallmarks of cancer. Cells 11, 2432, 2022.
]Search in Google Scholar
[
Wu F, Zhao J, Zhang E, Wu Q, Wu X, Zhang D, Liu Y, Wang R, Li W. Bisphenol A affects ovarian development in adolescent mice caused by genes expression change. Gene 740, 144535, 2020.
]Search in Google Scholar
[
Wu S, Zhang D, Zhang ZP, Soprano DR, Soprano KJ. Critical role of both retinoid nuclear receptors and retinoid-X-receptors in mediating growth inhibition of ovarian cancer cells by all-trans retinoic acid. Oncogene 17, 2839–2849, 1998.
]Search in Google Scholar
[
Ziv-Gal A, Flaws JA. Evidence for bisphenol A-induced female infertility: a review (2007–2016). Fertil Steril 106, 827–856, 2016.
]Search in Google Scholar
