This work is licensed under the Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License.
Mahid S.S., Colliver D.W., Crawford N.P., Martini B.D., Doll M.A., Hein D.W., Cobbs G.A., Petras R.E., Galandiuk S.: Characterization of N-acetyltransferase 1 and 2 polymorphisms and haplotype analysis for inflammatory bowel disease and sporadic colorectal carcinoma. BMC Med. Genet., 2007; 8: 28MahidS.S.ColliverD.W.CrawfordN.P.MartiniB.D.DollM.A.HeinD.W.CobbsG.A.PetrasR.E.GalandiukS.Characterization of N-acetyltransferase 1 and 2 polymorphisms and haplotype analysis for inflammatory bowel disease and sporadic colorectal carcinomaBMC Med. Genet.200782810.1186/1471-2350-8-28Search in Google Scholar
Rychlik-Sych M., Skrętkowicz J.: Metabolizm leków. Farm. Pol., 2008; 64: 51–60Rychlik-SychM.SkrętkowiczJ.Metabolizm lekówFarm. Pol.2008645160Search in Google Scholar
Sheehan D., Meade G., Foley V.M., Dowd C.A.: Structure, function and evolution of glutatione transferases: Implications for classification of non-mammalian members of an ancient enzyme super-family. Biochem. J., 2001; 360: 1–16SheehanD.MeadeG.FoleyV.M.DowdC.A.Structure, function and evolution of glutatione transferases: Implications for classification of non-mammalian members of an ancient enzyme super-familyBiochem. J.200136011610.1042/bj3600001Search in Google Scholar
Gallagher E.P., Gardner J.L., Barber D.S.: Several glutathione S-transferase isozymes that protect against injury are expressed in human liver mitochondria. Biochem. Pharmacol., 2006; 71: 1,619–1,628GallagherE.P.GardnerJ.L.BarberD.S.Several glutathione S-transferase isozymes that protect against injury are expressed in human liver mitochondriaBiochem. Pharmacol.2006711,6191,62810.1016/j.bcp.2006.02.018Search in Google Scholar
Marchewka Z., Piwowar A., Ruzik S., Długosz A.: Glutathione S-transferases class Pi and Mi and their significance in oncology. Postępy Hig. Med. Dośw., 2017; 71: 541–550MarchewkaZ.PiwowarA.RuzikS.DługoszA.Glutathione S-transferases class Pi and Mi and their significance in oncologyPostępy Hig. Med. Dośw.20177154155010.5604/01.3001.0010.3835Search in Google Scholar
Kerb R., Brockmöller J., Reum T., Roots I.: Deficiency of glutathi-one S-transferases T1 and M1 as heritable factors of increased cutaneous UV sensitivity. J. Invest. Dermatol., 1997; 108: 229–232KerbR.BrockmöllerJ.ReumT.RootsI.Deficiency of glutathi-one S-transferases T1 and M1 as heritable factors of increased cutaneous UV sensitivityJ. Invest. Dermatol.199710822923210.1111/1523-1747.ep12335337Search in Google Scholar
Strange R.C., Spiteri M.A., Ramachandran S., Fryer A.A.: Glutathione-S-transferase family of enzymes. Mutat. Res., 2001; 482: 21–26StrangeR.C.SpiteriM.A.RamachandranS.FryerA.A.Glutathione-S-transferase family of enzymesMutat. Res.2001482212610.1016/S0027-5107(01)00206-8Search in Google Scholar
Lo H.W., Ali-Osman F.: Genetic polymorphism and function of glutathione S-transferases in tumor drug resistance. Curr. Opin. Pharmacol., 2007; 7: 367–374LoH.W.Ali-OsmanF.Genetic polymorphism and function of glutathione S-transferases in tumor drug resistanceCurr. Opin. Pharmacol.2007736737410.1016/j.coph.2007.06.00917681492Search in Google Scholar
Matic M.G., Coric V.M., Savic-Radojevic A.R., Bulat P.V., Pljesa-Ercegovac M.S., Dragicevic D.P., Djukic T.I., Simic T.P., Pekmezovic T.D.: Does occupational exposure to solvents and pesticides in association with glutathione S-transferase A1, M1, P1, and T1 polymorphisms increase the risk of bladder cancer? The Belgrade Case-Control Study. PLoS One, 2014; 9: e99448MaticM.G.CoricV.M.Savic-RadojevicA.R.BulatP.V.Pljesa-ErcegovacM.S.DragicevicD.P.DjukicT.I.SimicT.P.PekmezovicT.D.Does occupational exposure to solvents and pesticides in association with glutathione S-transferase A1, M1, P1, and T1 polymorphisms increase the risk of bladder cancer? The Belgrade Case-Control StudyPLoS One20149e9944810.1371/journal.pone.0099448405177224914957Search in Google Scholar
Bolt H.M., Thier R.: Relevance of the deletion polymorphisms of the glutathione S-transferases GSTT1 and GSTM1 in pharmacology and toxicology. Curr. Drug Metab., 2006; 7: 613-628BoltH.M.ThierR.Relevance of the deletion polymorphisms of the glutathione S-transferases GSTT1 and GSTM1 in pharmacology and toxicologyCurr. Drug Metab.2006761362810.2174/13892000677801778616918316Search in Google Scholar
Board P.G., Menon D.: Glutathione transferases, regulators of cellular metabolism and physiology. Biochim. Biophys. Acta, 2013; 1830: 3,267–3,288BoardP.G.MenonD.Glutathione transferases, regulators of cellular metabolism and physiologyBiochim. Biophys. Acta201318303,2673,28810.1016/j.bbagen.2012.11.01923201197Search in Google Scholar
Fryer A.A., Bianco A., Hepple M., Jones P.W., Strange R.C., Spiteri M.A.: Polymorphism at the glutathione S-transferase GSTP1 locus. A new marker for bronchial hyperresponsiveness and asthma. Am. J. Respir. Crit. Care Med., 2000; 161: 1,437–1,442FryerA.A.BiancoA.HeppleM.JonesP.W.StrangeR.C.SpiteriM.A.Polymorphism at the glutathione S-transferase GSTP1 locus. A new marker for bronchial hyperresponsiveness and asthmaAm. J. Respir. Crit. Care Med.20001611,4371,44210.1164/ajrccm.161.5.9903006Search in Google Scholar
Dumitrescu R.G., Cotarla I.: Understanding breast cancer risk— where do we stand in 2005? J. Cell. Mol. Med., 2005; 9: 208–221DumitrescuR.G.CotarlaI.Understanding breast cancer risk— where do we stand in 2005?J. Cell. Mol. Med.2005920822110.1111/j.1582-4934.2005.tb00350.xSearch in Google Scholar
Jardim B.V., Moschetta M.G., Gelaleti G.B., Leonel C., Regiani V.R., de Santi Neto D., Bordin-Junior N.A., Perea S.A., Zuccari D.A.: Glutathione transferase pi (GSTpi) expression in breast cancer: An immunohistochemical and molecular study. Acta Histochem., 2012; 114: 510–517JardimB.V.MoschettaM.G.GelaletiG.B.LeonelC.RegianiV.R.de Santi NetoD.Bordin-JuniorN.A.PereaS.A.ZuccariD.A.Glutathione transferase pi (GSTpi) expression in breast cancer: An immunohistochemical and molecular studyActa Histochem.201211451051710.1016/j.acthis.2011.09.005Search in Google Scholar
Strange R.C., Fryer A.A.: The glutathione S-transferases: Influence of polymorphism on cancer susceptibility. IARC Sci. Publ., 1999; 148: 231–249StrangeR.C.FryerA.A.The glutathione S-transferases: Influence of polymorphism on cancer susceptibilityIARC Sci. Publ.199914823124910.1016/S0009-2797(97)00172-5Search in Google Scholar
Habdous M., Siest G., Herbeth B., Vincent-Viry M., Visvikis S.: Glutathione S-transferases genetic polymorphisms and human diseases: Overview of epidemiological studies. Ann. Biol. Clin., 2004; 62: 15–24HabdousM.SiestG.HerbethB.Vincent-ViryM.VisvikisS.Glutathione S-transferases genetic polymorphisms and human diseases: Overview of epidemiological studiesAnn. Biol. Clin.2004621524Search in Google Scholar
Pljesa-Ercegovac M., Savic-Radojevic A., Dragicevic D., Mimic-Oka J., Matic M., Sasic T., Pekmezovic T., Vuksanovic A., Simic T.: Enhanced GSTP1 expression in transitional cell carcinoma of urinary bladder is associated with altered apoptotic pathways. Urol. Oncol., 2011; 29: 70–77Pljesa-ErcegovacM.Savic-RadojevicA.DragicevicD.Mimic-OkaJ.MaticM.SasicT.PekmezovicT.VuksanovicA.SimicT.Enhanced GSTP1 expression in transitional cell carcinoma of urinary bladder is associated with altered apoptotic pathwaysUrol. Oncol.201129707710.1016/j.urolonc.2008.10.01919162514Search in Google Scholar
Lo Schiavo A., Ruocco E., Brancaccio G., Caccavale S., Ruocco V., Wolf R.: Bullous pemphigoid: Etiology, pathogenesis, and inducing factors: Facts and controversies. Clin. Dermatol., 2013; 31: 391–399Lo SchiavoA.RuoccoE.BrancaccioG.CaccavaleS.RuoccoV.WolfR.Bullous pemphigoid: Etiology, pathogenesis, and inducing factors: Facts and controversiesClin. Dermatol.20133139139910.1016/j.clindermatol.2013.01.00623806156Search in Google Scholar
Schmidt E., Reimer S., Kruse N., Jainta S., Bröcker E.B., Marinkovich M.P., Giudice G.J., Zillikens D.: Autoantibodies to BP180 associated with bullous pemphigoid release interleukin-6 and interleukin-8 from cultured human keratinocytes. J. Invest. Dermatol., 2000; 115: 842–848SchmidtE.ReimerS.KruseN.JaintaS.BröckerE.B.MarinkovichM.P.GiudiceG.J.ZillikensD.Autoantibodies to BP180 associated with bullous pemphigoid release interleukin-6 and interleukin-8 from cultured human keratinocytesJ. Invest. Dermatol.200011584284810.1046/j.1523-1747.2000.00141.x11069622Search in Google Scholar
Waszczykowska E., Wysoczańska K., Żebrowska A.: The role of cytokines in the pathogenesis of autoimmune bullous skin disease. Alerg. Astma Immun., 2004; 9: 179–186WaszczykowskaE.WysoczańskaK.ŻebrowskaA.The role of cytokines in the pathogenesis of autoimmune bullous skin diseaseAlerg. Astma Immun.20049179186Search in Google Scholar
Zakka L.R., Reche P., Ahmed A.R.: Role of MHC Class II genes in the pathogenesis of pemphigoid. Autoimmun. Rev., 2011; 11: 40–47ZakkaL.R.RecheP.AhmedA.R.Role of MHC Class II genes in the pathogenesis of pemphigoidAutoimmun. Rev.201111404710.1016/j.autrev.2011.07.00221782980Search in Google Scholar
de Graauw E., Sitaru C., Horn M.P., Borradori L., Yousefi S., Simon D., Simon H.U.: Monocytes enhance neutrophil-induced blister formation in an ex vivo model of bullous pemphigoid. Allergy, 2018; 73: 1,119–1,130de GraauwE.SitaruC.HornM.P.BorradoriL.YousefiS.SimonD.SimonH.U.Monocytes enhance neutrophil-induced blister formation in an ex vivo model of bullous pemphigoidAllergy2018731,1191,13010.1111/all.1337629222810Search in Google Scholar
Shah A.A., Sinha A.A.: Oxidative stress and autoimmune skin disease. Eur. J. Dermatol., 2013; 23: 5–13ShahA.A.SinhaA.A.Oxidative stress and autoimmune skin diseaseEur. J. Dermatol.20132351310.1684/ejd.2012.188423420016Search in Google Scholar
Otten J.V., Hashimoto T., Hertl M., Payne A.S., Sitaru C.: Molecular diagnosis in autoimmune skin blistering conditions. Curr. Mol. Med., 2014; 14: 69–95OttenJ.V.HashimotoT.HertlM.PayneA.S.SitaruC.Molecular diagnosis in autoimmune skin blistering conditionsCurr. Mol. Med.201414699510.2174/15665240113136660079390571624160488Search in Google Scholar
Gorukmez O., Yakut T., Gorukmez O., Sag S.O., Topak A., Sahinturk S., Kanat O.: Glutathione S-transferase T1, M1 and P1 genetic polymorphisms and susceptibility to colorectal cancer in Turkey. Asian Pac. J. Cancer Prev., 2016; 17: 3,855–3,859GorukmezO.YakutT.GorukmezO.SagS.O.TopakA.SahinturkS.KanatO.Glutathione S-transferase T1, M1 and P1 genetic polymorphisms and susceptibility to colorectal cancer in TurkeyAsian Pac. J. Cancer Prev.2016173,8553,859Search in Google Scholar
Abbas A., Delvinquiere K., Lechevrel M., Lebailly P., Gauduchon P., Launoy G., Sichel F.: GSTM1, GSTT1, GSTP1 and CYP1A1 genetic polymorphisms and susceptibility to esophageal cancer in a French population: Different pattern of squamous cell carcinoma and adenocarcinoma. World J. Gastroenterol., 2004; 10: 3,389–3,393AbbasA.DelvinquiereK.LechevrelM.LebaillyP.GauduchonP.LaunoyG.SichelF.GSTM1, GSTT1, GSTP1 and CYP1A1 genetic polymorphisms and susceptibility to esophageal cancer in a French population: Different pattern of squamous cell carcinoma and adenocarcinomaWorld J. Gastroenterol.2004103,3893,39310.3748/wjg.v10.i23.3389457621515526353Search in Google Scholar
Kulbacka J., Saczko J., Chwiłkowska A.: Oxidative stress in cells damage processes. Pol. Merk. Lekarski, 2009; 27: 44–47KulbackaJ.SaczkoJ.ChwiłkowskaA.Oxidative stress in cells damage processesPol. Merk. Lekarski2009274447Search in Google Scholar
Sheu S.S., Nauduri D., Anders M.W.: Targeting antioxidants to mitochondria: A new therapeutic direction. Biochim. Biophys. Acta, 2006; 1,762: 256–265SheuS.S.NauduriD.AndersM.W.Targeting antioxidants to mitochondria: A new therapeutic directionBiochim. Biophys. Acta20061,76225626510.1016/j.bbadis.2005.10.00716352423Search in Google Scholar
Tie D., Da X., Natsuga K., Yamada N., Yamamoto O., Morita E.: Bullous pemphigoid IgG induces cell dysfunction and enhances the motility of epidermal keratinocytes via Rac1/proteasome activation. Front. Immunol., 2019; 10: 200TieD.DaX.NatsugaK.YamadaN.YamamotoO.MoritaE.Bullous pemphigoid IgG induces cell dysfunction and enhances the motility of epidermal keratinocytes via Rac1/proteasome activationFront. Immunol.20191020010.3389/fimmu.2019.00200637934430809225Search in Google Scholar
Sarniak A., Lipińska J., Tytman K., Lipińska S.: Endogenous mechanisms of reactive oxygen species (ROS) generation. Postępy Hig. Med. Dośw., 2016; 70: 1,150–1,165SarniakA.LipińskaJ.TytmanK.LipińskaS.Endogenous mechanisms of reactive oxygen species (ROS) generationPostępy Hig. Med. Dośw.2016701,1501,16510.5604/17322693.122425927892899Search in Google Scholar
Bratic A., Larsson N.G.: The role of mitochondria in aging. J. Clin. Invest., 2013; 123: 951–957BraticA.LarssonN.G.The role of mitochondria in agingJ. Clin. Invest.201312395195710.1172/JCI64125358212723454757Search in Google Scholar
Wang C.H., Wu S.B., Wu Y.T., Wei Y.H.: Oxidative stress response elicited by mitochondrial dysfunction: Implication in the pathophysiology of aging. Exp. Biol. Med., 2013; 238: 450–460WangC.H.WuS.B.WuY.T.WeiY.H.Oxidative stress response elicited by mitochondrial dysfunction: Implication in the pathophysiology of agingExp. Biol. Med.201323845046010.1177/153537021349306923856898Search in Google Scholar
Maeshima E., Liang X.M., Goda M., Otani H., Mune M.: The efficacy of vitamin E against oxidative damage and autoantibody production in systemic lupus erythematosus: A preliminary study. Clin. Rheumatol., 2007; 26: 401–404MaeshimaE.LiangX.M.GodaM.OtaniH.MuneM.The efficacy of vitamin E against oxidative damage and autoantibody production in systemic lupus erythematosus: A preliminary studyClin. Rheumatol.20072640140410.1007/s10067-006-0477-x17143589Search in Google Scholar
Shah A.A., Dey-Rao R., Seiffert-Sinha K., Sinha A.A.: Increased oxidative stress in pemphigus vulgaris is related to disease activity and HLA-association. Autoimmunity, 2016; 49: 248–257ShahA.A.Dey-RaoR.Seiffert-SinhaK.SinhaA.A.Increased oxidative stress in pemphigus vulgaris is related to disease activity and HLA-associationAutoimmunity20164924825710.3109/08916934.2016.114567526911801Search in Google Scholar
Casciola-Rosen L., Rosen A.: Ultraviolet light-induced keratinocyte apoptosis: A potential mechanism for the induction of skin lesions and autoantibody production in LE. Lupus, 1997; 6: 175–180Casciola-RosenL.RosenA.Ultraviolet light-induced keratinocyte apoptosis: A potential mechanism for the induction of skin lesions and autoantibody production in LELupus1997617518010.1177/0961203397006002139061666Search in Google Scholar
Salimi S., Nakhaee A., Jafari M., Jahantigh D., Sandooghi M., Zakeri Z., Shahrakipour M., Naghavi A., Farajian-Mashhadi F.: Combination effect of GSTM1, GSTT1 and GSTP1 polymorphisms and risk of systemic lupus erythematosus. Iran. J. Public Health, 2015; 44: 814–821SalimiS.NakhaeeA.JafariM.JahantighD.SandooghiM.ZakeriZ.ShahrakipourM.NaghaviA.Farajian-MashhadiF.Combination effect of GSTM1, GSTT1 and GSTP1 polymorphisms and risk of systemic lupus erythematosusIran. J. Public Health201544814821Search in Google Scholar
Glesse N., Rohr P., Monticielo O.A., Rech T.F., Brenol J.C., Xavier R.M., Kvitko K., Chies J.A.: Genetic polymorphisms of glutathione S-transferases and cytochrome P450 enzymes as susceptibility factors to systemic lupus erythematosus in southern Brazilian patients. Mol. Biol. Rep., 2014; 41: 6,167–6,179GlesseN.RohrP.MonticieloO.A.RechT.F.BrenolJ.C.XavierR.M.KvitkoK.ChiesJ.A.Genetic polymorphisms of glutathione S-transferases and cytochrome P450 enzymes as susceptibility factors to systemic lupus erythematosus in southern Brazilian patientsMol. Biol. Rep.2014416,1676,17910.1007/s11033-014-3496-824981927Search in Google Scholar
Fraser P.A., Ding W.Z., Mohseni M., Treadwell E.L., Dooley M.A., St Clair E.W., Gilkeson G.S., Cooper G.S.: Glutathione S-transferase M null homozygosity and risk of systemic lupus erythematosus associated with sun exposure: A possible gene-environmental interaction for autoimmunity. J. Rheumatol., 2003; 30: 276–282FraserP.A.DingW.Z.MohseniM.TreadwellE.L.DooleyM.A.St ClairE.W.GilkesonG.S.CooperG.S.Glutathione S-transferase M null homozygosity and risk of systemic lupus erythematosus associated with sun exposure: A possible gene-environmental interaction for autoimmunityJ. Rheumatol.200330276282Search in Google Scholar
Karlson E.W., Watts J., Signorovitch J., Bonetti M., Wright E., Cooper G.S., McAlindon T.E., Costenbader K.H., Massarotti E.M., Fitzgerald L.M., et al.: Effect of glutathione S-transferase polymorphisms and proximity to hazardous waste sites on time to systemic lupus erythematosus diagnosis: Results From the Roxbury Lupus Project. Arthritis Rheum., 2007; 56: 244–254KarlsonE.W.WattsJ.SignorovitchJ.BonettiM.WrightE.CooperG.S.McAlindonT.E.CostenbaderK.H.MassarottiE.M.FitzgeraldL.M.Effect of glutathione S-transferase polymorphisms and proximity to hazardous waste sites on time to systemic lupus erythematosus diagnosis: Results From the Roxbury Lupus ProjectArthritis Rheum.20075624425410.1002/art.2230817195228Search in Google Scholar
Tew M.B., Reveille J.D., Arnett F.C., Friedman A.W., McNearney T., Fischbach M., Ahn C., Tan F.K.: Glutathione S-transferase genotypes in systemic sclerosis and their association with clinical manifestations in early disease. Genes Immun., 2001; 2: 236–238TewM.B.ReveilleJ.D.ArnettF.C.FriedmanA.W.McNearneyT.FischbachM.AhnC.TanF.K.Glutathione S-transferase genotypes in systemic sclerosis and their association with clinical manifestations in early diseaseGenes Immun.2001223623810.1038/sj.gene.636375611477481Search in Google Scholar
Palmer C.N., Young V., Ho M., Doney A., Belch J.J.: Association of common variation in glutathione S-transferase genes with premature development of cardiovascular disease in patients with systemic sclerosis. Arthritis Rheum., 2003; 48: 854–855PalmerC.N.YoungV.HoM.DoneyA.BelchJ.J.Association of common variation in glutathione S-transferase genes with premature development of cardiovascular disease in patients with systemic sclerosisArthritis Rheum.20034885485510.1002/art.1095512632442Search in Google Scholar